Bullseye tick bite on dog

Bullseye tick bite on dog

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Bullseye tick bite on dog's back (South Central Kansas). Dog was euthanized with the diagnosis of myocarditis, due to suspected rabies exposure

On December 13, 2015, a 4-year-old Boxer was evaluated at the Kansas State University Veterinary Teaching Hospital (KSU-VTH) for a tick bite. The dog had visited its family veterinarian and, while the dog's owners were working with the veterinarian in another room, the dog was scratched by a tick that remained attached in the hair of its back. The tick was successfully removed, but the dog began to scratch vigorously and soon developed an open wound over the site of the tick bite and a swollen area over the lower back. During the examination, the dog was alert and had no significant clinical abnormalities.

An ultrasound evaluation of the wound was performed to assess its depth and was unremarkable. Blood was collected for hematology and serum biochemistry testing, and cytology samples were taken.

When the dog's owners returned to the examination room, they explained that the dog was exhibiting signs of excitement. The dog began acting more alert, but the owners believed this to be the normal behavior of the dog after it was scratched by a tick and they had removed it. The dog, however, refused to lie down and was pacing in circles and whining. The owners also noted that the dog had begun to vomit frequently. The owners did not provide medication at this time. The dog became aggressive and began biting and scratching, and they called a veterinarian, who was able to sedate the dog with methadone hydrochloride (0.5 ,mg/kg subcutaneously) and midazolam (0.15 ,mg/kg intramuscularly). After the sedation, the dog's owners were unable to locate a blood-borne pathogen clinic in the area, and they were unable to afford to transport the dog for rabies testing.

One hour later, the dog became febrile (39.2 ,°C, 102.4 ,°F). The dog's behavior changed, as the dog was pacing, growling, and shaking, as well as trying to bite its own face. The owners gave a history of having observed this behavior before the event and stated that they had never seen this dog act this way before. At this time, the owners stated that the dog was experiencing vomiting, not urinating or defecating. The owners did not feed or water the dog.

Upon physical examination, the dog was lethargic and was not eating. It was unable to stand or walk and appeared to be having seizures. The dog was not able to swallow. The dog was unable to urinate, although the owners were not certain if this was related to the lack of appetite or if the dog was dehydrated. The dog had a mild respiratory rate, had a normal heart rate (122 beats/min), and a normal respiratory rate (22 breaths/min). The dog was panting, had a heart rate of 98 beats/min, and a temperature of 41.5 ,°C (106.8 ,°F). On abdominal palpation, there was no evidence of pain, pain with deep palpation of the area of the abdomen, or decreased bowel sounds. The dog did not respond to a painful stimulus.

The dog was hospitalized for a prolonged period. The owners continued to feed and water the dog, and they observed that the dog ate and drank. No vomiting or diarrhea occurred during this time.

Results of Complete Blood Count (CBC), Chemistry Panel, Urinalysis {#sec4}


CBC demonstrated a leukocytosis (17.7 × 10^9^ ,cells/L, reference range: 5--12 × 10^9^ ,cells/L). Chemistry panel revealed hyperbilirubinemia (2.65 ,mg/dL, reference range: <,1.00 ,mg/dL) and hyperbilirubinemia (direct, 2.73 ,mg/dL, reference range: <,0.70 ,mg/dL). No bilirubin was found in the urinalysis.

A plain radiograph of the lumbar spine revealed a large soft tissue opacity in the subcutaneous tissues of the lumbar spine and small to moderate soft tissue opacity throughout the rest of the spine. A computed tomography (CT) scan of the abdomen and thorax ([Fig 1](#fig1){ref-type="fig"}) revealed the presence of an extensive subcutaneous emphysema throughout the entire abdominal and thoracic cavity with associated fluid accumulations extending into the fascial planes of the abdominal wall, the thoracic wall, and the lungs, with minimal to moderate amounts of fascial air, fluid, and gas throughout the abdominal and thoracic cavity. Fluid accumulations were observed along the lumbar spine. There was no significant free air within the abdomen or thorax. There was a moderate amount of fat along the iliopsoas muscles. There was no evidence of pneumothorax. An exploratory laparotomy was performed for further evaluation of the findings on CT scan. During the exploratory laparotomy, the patient was noted to have small air-filled pockets of air in the omentum and left iliac lymph nodes.

After further resuscitation of the patient, she was transferred to the general medical service for a diagnostic evaluation and management of her pain. A magnetic resonance imaging of the lumbar spine was completed after transfer. Imaging revealed moderate to severe degenerative changes throughout the lumbar spine with the most prominent degenerative changes located at the L5-S1 disc space. There was extensive bilateral paravertebral air collections surrounding the right psoas major muscle, left psoas major muscle, iliac bone, and paraspinal muscles, extending laterally from the disc space at the L5-S1 level to the midline of the back at the L4-L5 level and from the left paravertebral muscles at the T11-T12 level to the midline of the back. The posterior elements were sclerotic and narrowed the canal. There were low-lying intradural air extensions through the soft tissues with extensive fascial involvement in the paraspinal and paravertebral areas. The L5 nerve roots were identified as having an intradural origin with direct extensions to the intradural air extensions. There were bilateral subarachnoid air collections in the conus medullaris, extending from the thoracolumbar junction to the level of the L5 nerve roots ([Fig. 1](#F1){ref-type="fig"}). The spinal cord compression caused a progressive weakness of both lower extremities.

At surgery, the epidural and intradural dissection revealed the air in the epidural space filling the extradural portion of the spinal canal and compressing the spinal cord. The spinal cord was swollen, pale, and pulpy. The T12 vertebral body was severely osteopenic with moderate bone loss and ankylosis of the L5 and L4 vertebral body. The L5 nerve roots, L5-S1 disc space, and left psoas major muscle were not visible. The L4 vertebral body was sclerotic with moderate to severe bone loss and ankylosis of the L5 and L4 vertebral body. There was severe lateral spinal stenosis with a bony spur present. The right psoas major muscle was edematous, and there was no muscle fibers visible. The right psoas major muscle was completely replaced by fat. The left pso

Watch the video: How to Treat Tick Bites Naturally - Tick Bites Natural Cure (May 2022).

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